Question 201 ptsHow is the slow after-hyperpolarisation (sAHP) generated in lamprey spinal neurons?Group of answer choicesit is due to the slow closure of the delayed rectifier channels opened during the downstroke of the action potentialit is mediated by calcium entering the cell which activates a slow calcium-activated potassium currentit is due to opening of sodium-activated calcium pumps in the nerve cell membraneit is due to activation of the Na+/K+ ATPase (sodium/potassium ATPase)it is due to an inhibitory post-synaptic input mediated by glycine Previous
Question
Question 201 ptsHow is the slow after-hyperpolarisation (sAHP) generated in lamprey spinal neurons?Group of answer choicesit is due to the slow closure of the delayed rectifier channels opened during the downstroke of the action potentialit is mediated by calcium entering the cell which activates a slow calcium-activated potassium currentit is due to opening of sodium-activated calcium pumps in the nerve cell membraneit is due to activation of the Na+/K+ ATPase (sodium/potassium ATPase)it is due to an inhibitory post-synaptic input mediated by glycine Previous
Solution
The slow after-hyperpolarisation (sAHP) in lamprey spinal neurons is generated through the process of calcium entering the cell, which activates a slow calcium-activated potassium current. This process is crucial for the regulation of neuronal excitability and the repolarization of the neuron after an action potential.
Here are the steps:
-
An action potential, or nerve impulse, is initiated in the neuron. This causes voltage-gated calcium channels to open.
-
Calcium ions flow into the neuron due to the concentration gradient (there is a higher concentration of calcium ions outside the neuron than inside).
-
The influx of calcium ions activates a type of potassium channel known as a calcium-activated potassium channel.
-
The activation of these channels allows potassium ions to flow out of the neuron, again due to the concentration gradient (there is a higher concentration of potassium ions inside the neuron than outside).
-
The outflow of potassium ions causes the neuron to become more negative, or hyperpolarized. This is the slow after-hyperpolarisation (sAHP).
-
The sAHP makes the neuron less likely to fire another action potential immediately, effectively providing a refractory period that helps to regulate the rate of nerve impulses.
So, the correct answer to your question is: "it is mediated by calcium entering the cell which activates a slow calcium-activated potassium current".
Similar Questions
Question 61 ptsThe slow after-hyperpolarisations (sAHPs) in some spinal neurons of the lamprey:Group of answer choicescontrol the speed of swimmingcontrol the direction of swimmingcontrol the alternation of contractions between the two sidescontrol the sensory inputscontrol the propagation of excitation along the spinal cord
Question 181 ptsIf a given neuropeptide decreases the amplitude of the slow after-hyperpolarisation (sAHP) in spinal neurons, its effect on lamprey swimming would be:Group of answer choicesbursts of muscle action potentials would be longer and the lamprey would have a slower speed of swimmingbursts of muscle action potentials would be shorter and the lamprey would have a slower speed of swimmingbursts of muscle action potentials would be longer and the lamprey would have a faster speed of swimmingmuscles on both sides of the lamprey would contract simultaneouslymuscle action potentials would fire at a lower frequency within bursts PreviousNext
Question 71 ptsRelease of endogenous serotonin (5-HT) in the lamprey spinal cord leads to:Group of answer choicespartial inhibition of action potential generation in motor neuronsinhibition of calcium-dependent potassium channels in the conditional pacemaker neuronsstimulation of the calcium-dependent potassium channels in the conditional pacemaker neuronsan increase in the frequency of cyclic oscillations of the spinal locomotor networkinhibition of NMDA-receptors on spinal neurons PreviousNext
Question 141 ptsIntracellular recordings from conditional pacemaker neurons in isolated lamprey spinal cord/brainstem preparations show:Group of answer choicesspontaneous action potentials triggered at resting membrane potentialslow oscillations of membrane potential with superimposed fast action potentialsslow oscillations of membrane potential in the absence of any neural or pharmacological excitationserotonin-mediated inhibitory synaptic potentialsslow oscillations driven by GABA-A mediated synaptic input with superimposed action potentials PreviousNext
Oscillations in the conditional pacemakers in the lamprey spinal cord involve Chloride channels and sodium channels Calcium channels and barium channels Potassium channels and barium channels Sodium channels and calcium channels Potassium channels and chloride channels
Upgrade your grade with Knowee
Get personalized homework help. Review tough concepts in more detail, or go deeper into your topic by exploring other relevant questions.